antibody against s1p2 (Proteintech)
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Antibody Against S1p2, supplied by Proteintech, used in various techniques. Bioz Stars score: 94/100, based on 77 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+s1pr2/S1PR2+Antibody/pm41701442-39-1-7
Average 94 stars, based on 77 article reviews
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Blocking Assay:Article Title: Overexpression of endothelial S1pr2 promotes blood-brain barrier disruption via JNK/c-Jun/MMP-9 pathway after traumatic brain injury in both in vivo and in vitro models. Article Snippet: .. After blocking with 5% non-fat milk, the membranes underwent incubation with primary antibodies targeting Erk: anti-Erk and antiphosphorylated Erk (1:1,000; #4370/#4695, CST), JNK: anti-JNK and anti-phosphorylated JNK (1:1,000; #9251/#9252, CST), p38: anti-p38 and anti-phosphorylated p38 (1:1,000; #9212/#9211, CST), c-Jun: Anti-c-Jun and anti-phosphorylated-c-Jun (1:1,000; #9165/ #2361), anti-MMP-9 (1:1,000; ab76003; Abcam), Article Title: Overexpression of endothelial S1pr2 promotes blood–brain barrier disruption via JNK/c-Jun/MMP-9 pathway after traumatic brain injury in both in vivo and in vitro models Article Snippet: .. After blocking with 5% non-fat milk, the membranes underwent incubation with primary antibodies targeting Erk: anti-Erk and anti-phosphorylated Erk (1:1,000; #4370/#4695, CST), JNK: anti-JNK and anti-phosphorylated JNK (1:1,000; #9251/#9252, CST), p38: anti-p38 and anti-phosphorylated p38 (1:1,000; #9212/#9211, CST), c-Jun: Anti-c-Jun and anti-phosphorylated-c-Jun (1:1,000; #9165/#2361), anti-MMP-9 (1:1,000; ab76003; Abcam), Incubation:Article Title: Overexpression of endothelial S1pr2 promotes blood-brain barrier disruption via JNK/c-Jun/MMP-9 pathway after traumatic brain injury in both in vivo and in vitro models. Article Snippet: .. After blocking with 5% non-fat milk, the membranes underwent incubation with primary antibodies targeting Erk: anti-Erk and antiphosphorylated Erk (1:1,000; #4370/#4695, CST), JNK: anti-JNK and anti-phosphorylated JNK (1:1,000; #9251/#9252, CST), p38: anti-p38 and anti-phosphorylated p38 (1:1,000; #9212/#9211, CST), c-Jun: Anti-c-Jun and anti-phosphorylated-c-Jun (1:1,000; #9165/ #2361), anti-MMP-9 (1:1,000; ab76003; Abcam), Article Title: VSV infection and LPS treatment alter serum bile acid profiles, bile acid biosynthesis, and bile acid receptors in mice Article Snippet: .. Deparaffinized specimens were incubated with fresh 0.3% hydrogen peroxide and then incubated with anti-FXR (25055-1-AP, 1:300, Proteintech, China), anti-TGR5 (ab72608, 1:300, abcam, UK), Article Title: Overexpression of endothelial S1pr2 promotes blood–brain barrier disruption via JNK/c-Jun/MMP-9 pathway after traumatic brain injury in both in vivo and in vitro models Article Snippet: .. After blocking with 5% non-fat milk, the membranes underwent incubation with primary antibodies targeting Erk: anti-Erk and anti-phosphorylated Erk (1:1,000; #4370/#4695, CST), JNK: anti-JNK and anti-phosphorylated JNK (1:1,000; #9251/#9252, CST), p38: anti-p38 and anti-phosphorylated p38 (1:1,000; #9212/#9211, CST), c-Jun: Anti-c-Jun and anti-phosphorylated-c-Jun (1:1,000; #9165/#2361), anti-MMP-9 (1:1,000; ab76003; Abcam), Article Title: VSV infection and LPS treatment alter serum bile acid profiles, bile acid biosynthesis, and bile acid receptors in mice. Article Snippet: .. Deparaffinized specimens were incubated with fresh 0.3% hydrogen peroxide and then incubated with anti-FXR (25055-1-AP, 1:300, Proteintech, China), anti-TGR5 (ab72608, 1:300, abcam, UK), other:Article Title: Fractional carbon dioxide laser-induced photothermal activation of mesenchymal stem cell-derived exosomes accelerates diabetic wound healing by enhancing angiogenesis Article Snippet: Article Title: Exogenous S1P via S1P receptor 2 induces CTGF expression through Src-RhoA-ROCK-YAP pathway in hepatic stellate cells. Article Snippet: 1 Department of Health Science and Technology, SAIHST, Sungkyunkwan University, 81 Irwon-Ro, Gangnam-Gu, Seoul 06351, South Korea 2 Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, 81 IrwonRo, Gangnam-Gu, Seoul 06351, South Korea 3 Samsung Medical Center, 81 IrwonRo, Gangnam-Gu, Seoul 06351, South Korea Abstract Background Hepatic fibrosis, a prevalent chronic liver condition, involves excessive extracellular matrix production associated with aberrant wound healing.. Hepatic stellate cells (HSCs) play a pivotal role in liver fibrosis, activated by inflammatory factors such as sphingosine 1-phosphate (S1P).. Despite S1P’s involvement in fibrosis, its specific role and downstream pathway in HSCs remain controversial. |
